Can ErbB2 Overexpression Protect Against Doxorubicin Cardiotoxicity?

Authors

Petra Rocic, New York Medical CollegeFollow

First Page

1235

Last Page

1236

Document Type

Response or Comment

Publication Date

10-1-2015

Department

Pharmacology

Keywords

Animals, Antineoplastic Agents, Antioxidants, Doxorubicin, Glutathione Peroxidase, Heart Diseases, Mitochondria, Heart, Myocytes, Cardiac, Oxidative Stress, Reactive Oxygen Species, Receptor, ErbB-2

Disciplines

Cardiology | Circulatory and Respiratory Physiology

Abstract

Comments

Comment on

ErbB2 overexpression upregulates antioxidant enzymes, reduces basal levels of reactive oxygen species, and protects against doxorubicin cardiotoxicity. [Am J Physiol Heart Circ Physiol. 2015]

Recommended Citation

Rocic, P. (2015). Can ErbB2 overexpression protect against doxorubicin cardiotoxicity? American Journal of Physiology.Heart and Circulatory Physiology, 309(8), H1235-6. doi:10.1152/ajpheart.00647.2015

Publisher's Statement

Originally published in American Journal of Physiology.Heart and Circulatory Physiology. https://doi.org/10.1152/ajpheart.00647.2015